CLEC5A is a critical receptor in innate immunity against Listeria infection-Reference-Cited by-同舟云学术

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CLEC5A is a critical receptor in innate immunity against Listeria infection

Published:2017-08-21 Issue:1 Volume:8 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Chen Szu-Ting,Li Fei-Ju,Hsu Tzy-yun,Liang Shu-Mei,Yeh Yi-Chen,Liao Wen-Yu,Chou Teh-Ying,Chen Nien-Jun,Hsiao Michael,Yang Wen-Bin,Hsieh Shie-Liang

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-017-00356-3.pdf

Reference52 articles.

1. Bakker, A. B., Baker, E., Sutherland, G. R., Phillips, J. H. & Lanier, L. L. Myeloid DAP12-associating lectin (MDL)-1 is a cell surface receptor involved in the activation of myeloid cells. Proc. Natl Acad. Sci. USA 96, 9792–9796 (1999).

2. Brown, G. D. et al. Dectin-1 is a major beta-glucan receptor on macrophages. J. Exp. Med. 196, 407–412 (2002).

3. Taylor, P. R. et al. Dectin-1 is required for beta-glucan recognition and control of fungal infection. Nat. Immunol. 8, 31–38 (2007).

4. Chen, S. T. et al. CLEC5A is critical for dengue-virus-induced lethal disease. Nature 453, 672–676 (2008).

5. Chen, S. T. et al. CLEC5A regulates Japanese encephalitis virus-induced neuroinflammation and lethality. PLoS. Pathog. 8, e1002655 (2012).

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