PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation-Reference-Cited by-同舟云学术

PIM kinases facilitate lentiviral evasion from SAMHD1 restriction via Vpx phosphorylation

Published:2019-04-23 Issue:1 Volume:10 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Miyakawa Kei,Matsunaga Satoko,Yokoyama Masaru^ORCID,Nomaguchi Masako,Kimura Yayoi,Nishi Mayuko,Kimura Hirokazu,Sato Hironori,Hirano Hisashi,Tamura Tomohiko,Akari Hirofumi,Miura Tomoyuki,Adachi Akio,Sawasaki Tatsuya,Yamamoto Naoki,Ryo Akihide

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-019-09867-7.pdf

Reference50 articles.

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2. Brito, A. F. & Pinney, J. W. Protein-protein interactions in virus-host systems. Front. Microbiol. 8, 1557 (2017).

3. Kirchhoff, F. Immune evasion and counteraction of restriction factors by HIV-1 and other primate lentiviruses. Cell. Host. Microbe 8, 55–67 (2010).

4. Goldstone, D. C. et al. HIV-1 restriction factor SAMHD1 is a deoxynucleoside triphosphate triphosphohydrolase. Nature 480, 379–382 (2011).

5. Lahouassa, H. et al. SAMHD1 restricts the replication of human immunodeficiency virus type 1 by depleting the intracellular pool of deoxynucleoside triphosphates. Nat. Immunol. 13, 223–228 (2012).

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