Endosomal NOX2 oxidase exacerbates virus pathogenicity and is a target for antiviral therapy
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/s41467-017-00057-x.pdf
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3. Imai, Y. et al. Identification of oxidative stress and Toll-like receptor 4 signaling as a key pathway of acute lung injury. Cell 133, 235–249 (2008).
4. Snelgrove, R. J., Edwards, L., Rae, A. J. & Hussell, T. An absence of reactive oxygen species improves the resolution of lung influenza infection. Eur. J. Immunol. 36, 1364–1373 (2006).
5. To, E. E., Broughton, B. R., Hendricks, K. S., Vlahos, R. & Selemidis, S. Influenza A virus and TLR7 activation potentiate NOX2 oxidase-dependent ROS production in macrophages. Free Radic. Res. 48, 940–947 (2014).
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