Gimap5-dependent inactivation of GSK3β is required for CD4+ T cell homeostasis and prevention of immune pathology-Reference-Cited by-同舟云学术

Gimap5-dependent inactivation of GSK3β is required for CD4+ T cell homeostasis and prevention of immune pathology

Published:2018-01-30 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Patterson Andrew R.^ORCID,Endale Mehari^ORCID,Lampe Kristin,Aksoylar Halil I.,Flagg Aron,Woodgett Jim R.^ORCID,Hildeman David^ORCID,Jordan Michael B.,Singh Harinder,Kucuk Zeynep,Bleesing Jack,Hoebe Kasper

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-02897-7.pdf

Reference74 articles.

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3. Shin, J. H. et al. IA-2 autoantibodies in incident type I diabetes patients are associated with a polyadenylation signal polymorphism in GIMAP5. Genes Immun. 8, 503–512 (2007).

4. Heinonen, M. T. et al. GIMAP GTPase family genes: potential modifiers in autoimmune diabetes, asthma, and allergy. J. Immunol. 194, 5885–5894 (2015).

5. Aksoylar, H. I., Lampe, K., Barnes, M. J., Plas, D. R. & Hoebe, K. Loss of immunological tolerance in Gimap5-deficient mice is associated with loss of Foxo in CD4+T Cells. J. Immunol. 188, 146–154 (2012).

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