HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130-Reference-Cited by-同舟云学术

HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130

Published:2018-02-22 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Göder Anja,Emmerich Claudia,Nikolova Teodora,Kiweler Nicole,Schreiber Maria,Kühl Toni,Imhof Diana^ORCID,Christmann Markus,Heinzel Thorsten,Schneider Günter,Krämer Oliver H.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-03096-0.pdf

Reference70 articles.

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3. Cimprich, K. A. & Cortez, D. ATR: an essential regulator of genome integrity. Nat. Rev. Mol. Cell Biol. 9, 616–627 (2008).

4. López-Contreras, A. J. & Fernandez-Capetillo, O. The ATR barrier to replication-born DNA damage. DNA Rep. 9, 1249–1255 (2010).

5. Sørensen, C. S. & Syljuasen, R. G. Safeguarding genome integrity: the checkpoint kinases ATR, CHK1 and WEE1 restrain CDK activity during normal DNA replication. Nucleic Acids Res. 40, 477–486 (2012).

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