A high-throughput screen indicates gemcitabine and JAK inhibitors may be useful for treating pediatric AML
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/s41467-019-09917-0.pdf
Reference61 articles.
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2. Rubnitz, J. E. & Inaba, H. Childhood acute myeloid leukaemia. Br. J. Haematol. 159, 259–276 (2012).
3. Athale, U. H. et al. Biology and outcome of childhood acute megakaryoblastic leukemia: a single institution’s experience. Blood 97, 3727–3732 (2001).
4. Barnard, D. R. et al. Comparison of childhood myelodysplastic syndrome, AML FAB M6 or M7, CCG 2891: report from the Children’s Oncology Group. Pediatr. Blood Cancer 49, 17–22 (2007).
5. Chen, Y. et al. Prognostic significance of 11q23 aberrations in adult acute myeloid leukemia and the role of allogeneic stem cell transplantation. Leukemia 27, 836–842 (2013).
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