Smu1 and RED are required for activation of spliceosomal B complexes assembled on short introns
Author:
Funder
Deutsche Forschungsgemeinschaft
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/s41467-019-11293-8.pdf
Reference57 articles.
1. Wahl, M. C., Will, C. L. & Lührmann, R. The spliceosome: design principles of a dynamic RNP machine. Cell 136, 701–718 (2009).
2. Will, C. L. & Lührmann, R. Spliceosome structure and function. Cold Spring Harb. Perspect. Biol. 3, a003707 (2011).
3. Hube, F. & Francastel, C. Mammalian introns: when the junk generates molecular diversity. Int. J. Mol. Sci. 16, 4429–4452 (2015).
4. Ruskin, B., Greene, J. M. & Green, M. R. Cryptic branch point activation allows accurate in vitro splicing of human beta-globin intron mutants. Cell 41, 833–844 (1985).
5. Wieringa, B., Hofer, E. & Weissmann, C. A minimal intron length but no specific internal sequence is required for splicing the large rabbit beta-globin intron. Cell 37, 915–925 (1984).
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