GCN5L1 modulates cross-talk between mitochondria and cell signaling to regulate FoxO1 stability and gluconeogenesis
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/s41467-017-00521-8.pdf
Reference39 articles.
1. Scott, I., Webster, B. R., Li, J. H. & Sack, M. N. Identification of a molecular component of the mitochondrial acetyltransferase programme: a novel role for GCN5L1. Biochem. J. 443, 655–661 (2012).
2. Zhang, A. et al. Biogenesis of lysosome-related organelles complex-1 subunit 1 (BLOS1) interacts with sorting nexin 2 and the endosomal sorting complex required for transport-I (ESCRT-I) component TSG101 to mediate the sorting of epidermal growth factor receptor into endosomal compartments. J. Biol. Chem. 289, 29180–29194 (2014).
3. Webster, B. R. et al. Restricted mitochondrial protein acetylation initiates mitochondrial autophagy. J. Cell. Sci. 126, 4843–4849 (2013).
4. Scott, I. et al. GCN5-like protein 1 (GCN5L1) controls mitochondrial content through coordinated regulation of mitochondrial biogenesis and mitophagy. J. Biol. Chem. 289, 2864–2872 (2014).
5. Webster, B. R., Scott, I., Traba, J., Han, K. & Sack, M. N. Regulation of autophagy and mitophagy by nutrient availability and acetylation. Biochim. Biophys. Acta. 1841, 525–534 (2014).
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