Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation-Reference-Cited by-同舟云学术

Hectd3 promotes pathogenic Th17 lineage through Stat3 activation and Malt1 signaling in neuroinflammation

Published:2019-02-11 Issue:1 Volume:10 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Cho Jonathan J.,Xu Zhiwei,Parthasarathy Upasana,Drashansky Theodore T.,Helm Eric Y.,Zuniga Ashley N.,Lorentsen Kyle J.,Mansouri Samira,Cho Joshua Y.,Edelmann Mariola J.^ORCID,Duong Duc M.,Gehring Torben,Seeholzer Thomas,Krappmann Daniel,Uddin Mohammad N.,Califano Danielle,Wang Rejean L.,Jin Lei,Li Hongmin^ORCID,Lv Dongwen,Zhou Daohong,Zhou Liang,Avram Dorina

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-019-08605-3.pdf

Reference70 articles.

1. Zúñiga, L. A., Jain, R., Haines, C. & Cua, D. J. Th17 cell development: from the cradle to the grave. Immunol. Rev. 252, 78–88 (2013).

2. Langrish, C. L. et al. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation. J. Exp. Med. 201, 233–240 (2005).

3. Park, H. et al. A distinct lineage of CD4 T cells regulates tissue inflammation by producing interleukin 17. Nat. Immunol. 6, 1133–1141 (2005).

4. Tzartos, J. S. et al. Interleukin-17 production in central nervous system-infiltrating T cells and glial cells is associated with active disease in multiple sclerosis. Am. J. Pathol. 172, 146–155 (2008).

5. Kebir, H. et al. Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation. Nat. Med. 13, 1173–1175 (2007).

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