Cardiac specific PRMT1 ablation causes heart failure through CaMKII dysregulation-Reference-Cited by-同舟云学术

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Cardiac specific PRMT1 ablation causes heart failure through CaMKII dysregulation

Published:2018-11-30 Issue:1 Volume:9 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Pyun Jung-Hoon,Kim Hyun-Ji,Jeong Myong-Ho,Ahn Byeong-Yun,Vuong Tuan Anh,Lee Dong I.,Choi Seri,Koo Seung-Hoi,Cho Hana,Kang Jong-Sun

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/s41467-018-07606-y.pdf

Reference56 articles.

1. Zhang, T. et al. The delta(C) isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure. Circ. Res. 92, 912–919 (2003).

2. Anderson, M. E., Brown, J. H. & Bers, D. M. CaMKII in myocardial hypertrophy and heart failure. J. Mol. Cell. Cardiol. 51, 468–473 (2011).

3. Hoch, B., Meyer, R., Hetzer, P., Krause, E. G. & Karczewski, P. Identification and expression of delta-isoforms of the multifunctional Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human myocardium. Circ. Res. 84, 713–721 (1999).

4. Little, G. H. et al. Critical role of nuclear calcium/calmodulin-dependent protein kinase II delta B in cardiomyocyte survival in cardiomyopathy. J. Biol. Chem. 284, 24857–24868 (2009).

5. Netticadan, T., Temsah, R. M., Kawabata, K. & Dhalla, N. S. Sarcoplasmic reticulum Ca2+/calmodulin-dependent protein kinase is altered in heart failure. Circ. Res. 86, 596–605 (2000).

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