The role of C5a in acute lung injury induced by highly pathogenic viral infections
Author:
Affiliation:
1. Laboratory of Immunology, Institute of Basic Medical Sciences, Beijing 100850, China
2. Department of Pathology, University of Michigan, Ann Arbor, Michigan 48109, USA
3. InflaRx GmbH, Jena 07745, Germany
Publisher
Informa UK Limited
Subject
Virology,Infectious Diseases,Drug Discovery,General Medicine,Immunology,Microbiology,Parasitology,Epidemiology
Link
http://www.nature.com/articles/emi201528.pdf
Reference104 articles.
1. Complement biosynthesis by human bronchoalveolar macrophages
2. Pulmonary alveolar type II epithelial cells synthesize and secrete proteins of the classical and alternative complement pathways.
3. Complement C5 Activation during Influenza A Infection in Mice Contributes to Neutrophil Recruitment and Lung Injury
4. Inhibition of Complement Activation Alleviates Acute Lung Injury Induced by Highly Pathogenic Avian Influenza H5N1 Virus Infection
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