Amnionless, essential for mouse gastrulation, is mutated in recessive hereditary megaloblastic anemia-Reference-Cited by-同舟云学术

Amnionless, essential for mouse gastrulation, is mutated in recessive hereditary megaloblastic anemia

Published:2003-02-18 Issue:3 Volume:33 Page:426-429
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Tanner Stephan M.,Aminoff Maria,Wright Fred A.,Liyanarachchi Sandya,Kuronen Mervi,Saarinen Anne,Massika Orit,Mandel Hanna,Broch Harald,de la Chapelle Albert

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng1098.pdf

Reference23 articles.

1. Kalantry, S. et al. The amnionless gene, essential for mouse gastrulation, encodes a visceral-endoderm–specific protein with an extracellular cysteine-rich domain. Nat. Genet. 27, 412–416 (2001).

2. Wang, X. et al. A candidate gene for the amnionless gastrulation stage mouse mutation encodes a TRAF-related protein. Dev. Biol. 177, 274–290 (1996).

3. Tomihara-Newberger, C. et al. The amn gene product is required in extraembryonic tissues for the generation of middle primitive streak derivatives. Dev. Biol. 204, 34–54 (1998).

4. Dunn, N.R. & Hogan, B.L. How does the mouse get its trunk? Nat. Genet. 27, 351–352 (2001).

5. Imerslund, O. Idiopathic chronic megaloblastic anemia in children. Acta Paediatr. Scand. Suppl, 1–115 (1960).

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