Is calcium the 'cure' for dilated cardiomyopathy?
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Biochemistry, Genetics and Molecular Biology,General Medicine
Link
http://www.nature.com/articles/nm1299_1353.pdf
Reference6 articles.
1. Minamisawa, S. et al. Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy. Cell 99, 313–322 (1999).
2. Koss, K.L. & Kranias, E.G. Phospholamban: a prominent regulator of myocardial contractility. Circ. Res. 79, 1059–1063 (1996).
3. Palmiter, K.A. & Solaro, R.J. Molecular mechanisms regulating the myofilament response to Ca2+: implications of mutations causal for familial hypertrophic cardiomyopathy. Basic Res. Cardiol. 92, 63–74 (1997).
4. Chicurel, M.E., Singer, R.H., Meyer, C.J. & Ingber, D.E. Integrin binding and mechanical tension induce movement of mRNA and ribosomes to focal adhesions. Nature 392, 730–733 (1998).
5. Rockman, H.A. et al. Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice. Proc. Natl. Acad. Sci. USA 95, 7000–7005 (1998).
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