Complete hydatidiform mole retaining a chromosome 11 of maternal origin: molecular genetic analysis of a case-Reference-Cited by-同舟云学术

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Complete hydatidiform mole retaining a chromosome 11 of maternal origin: molecular genetic analysis of a case

Published:2004-08-16 Issue:9 Volume:17 Page:1155-1160
ISSN:0893-3952
Container-title:Modern Pathology
language:en
Short-container-title:Mod Pathol

Author:

Fisher Rosemary A,Nucci Marisa R,Thaker Harshwardhan M,Weremowicz Stanislawa,Genest David R,Castrillon Diego H

Publisher

Springer Science and Business Media LLC

Subject

Pathology and Forensic Medicine

Link

http://www.nature.com/articles/3800175.pdf

Reference15 articles.

1. Redline RW, Abdul-Karim FW . Pathology of gestational trophoblastic disease. Semin Oncol 1995;22:96–108.

2. Zaragoza MV, Surti U, Redline RW, et al. Parental origin and phenotype of triploidy in spontaneous abortions: predominance of diandry and association with the partial hydatidiform mole. Am J Hum Genet 2000;66:1807–1820.

3. Castrillon DH, Sun D, Weremowicz S, et al. Discrimination of complete hydatidiform mole from its mimics by immunohistochemistry of the paternally imprinted gene product p57KIP2. Am J Surg Pathol 2001;25:1225–1230.

4. Fisher RA, Hodges MD, Rees HC, et al. The maternally transcribed gene p57KIP2 (CDNK1C) is abnormally expressed in both androgenetic and biparental complete hydatidiform moles. Hum Mol Genet 2002;11:1–6.

5. Nucci MR, Castrillon DH, Bai H, et al. Biomarkers in diagnostic obstetric and gynecologic pathology: a review. Adv Anat Pathol 2003;10:55–68.

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