Expression levels of DNA methyltransferase genes do not correlate with p15INK4B gene methylation in myelodysplastic syndromes
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/2403046.pdf
Reference10 articles.
1. Uchida T, Kinoshita T, Nagai H, Nakahara Y, Saito H, Hotta T et al. Hypermethylation of the p15INK4B gene in myelodysplastic syndromes. Blood 1997; 90: 1403–1409.
2. Aoki E, Uchida T, Ohashi H, Nagai H, Murase T, Ichikawa A et al. Methylation status of the p15INK4B gene in hematopoietic progenitors and peripheral blood cells in myelodysplastic syndromes. Leukemia 2000; 14: 586–593.
3. Quesnel B, Guillerm G, Vereecque R, Wattel E, Preudhomme C, Bauters F et al. Methylation of the p15 (INK4b) gene in myelodysplastic syndrome is frequent and acquired during disease progression. Blood 1998; 91: 2985–2990.
4. Kautiainen TL, Jones PA . DNA methyltransferase levels in tumorigenic and nontumorigenic cells in culture. J Biol Chem 1986; 261: 1594–1598.
5. el-Deiry WS, Nelkin BD, Celano P, Yen RW, Falco JP, Hamilton SR et al. High expression of the DNA methyltransferase gene characterizes human neoplastic cells and progression stages of colon cancer. Proc Nat Acad Sci USA 1991; 88: 3470–3474.
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