Pim2 complements Flt3 wild-type receptor in hematopoietic progenitor cell transformation-Reference-Cited by-同舟云学术

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Pim2 complements Flt3 wild-type receptor in hematopoietic progenitor cell transformation

Published:2007-10-18 Issue:1 Volume:22 Page:78-86
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Agrawal S,Koschmieder S,Bäumer N,Reddy N G P,Berdel W E,Müller-Tidow C,Serve H

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2404988.pdf

Reference35 articles.

1. Stirewalt DL, Radich JP . The role of FLT3 in haematopoietic malignancies. Nat Rev Cancer 2003; 3: 650–665.

2. Mizuki M, Fenski R, Halfter H, Matsumura I, Schmidt R, Muller C et al. Flt3 mutations from patients with acute myeloid leukemia induce transformation of 32D cells mediated by the Ras and STAT5 pathways. Blood 2000; 96: 3907–3914.

3. Hayakawa F, Towatari M, Kiyoi H, Tanimoto M, Kitamura T, Saito H et al. Tandem-duplicated Flt3 constitutively activates STAT5 and MAP kinase and introduces autonomous cell growth in IL-3-dependent cell lines. Oncogene 2000; 19: 624–631.

4. Mizuki M, Schwable J, Steur C, Choudhary C, Agrawal S, Sargin B et al. Suppression of myeloid transcription factors and induction of STAT response genes by AML-specific Flt3 mutations. Blood 2003; 101: 3164–3173.

5. Allen JD, Verhoeven E, Domen J, van der Valk M, Berns A . Pim-2 transgene induces lymphoid tumors, exhibiting potent synergy with c-myc. Oncogene 1997; 15: 1133–1141.

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3. PIM2 regulates stemness through phosphorylation of 4E-BP1;Science Bulletin;2017-05

4. FLT3 inhibitors: clinical potential in acute myeloid leukemia;OncoTargets and Therapy;2017-02

5. Characterization of HJ-PI01 as a novel Pim-2 inhibitor that induces apoptosis and autophagic cell death in triple-negative human breast cancer;Acta Pharmacologica Sinica;2016-07-11

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