Further evidence that germline CEBPA mutations cause dominant inheritance of acute myeloid leukaemia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/2403788.pdf
Reference6 articles.
1. Smith ML, Cavenagh JD, Lister TA, Fitzgibbon J . Mutation of CEBPA in familial acute myeloid leukemia. N Engl J Med 2004; 351: 2403–2407.
2. Radomska HS, Huettner CS, Zhang P, Cheng T, Scadden DT, Tenen DG . CCAAT/enhancer binding protein alpha is a regulatory switch sufficient for induction of granulocytic development from bipotential myeloid progenitors. Mol Cell Biol 1998; 18: 4301–4314.
3. De Lord C, Powles R, Mehta J, Wilson K, Treleaven J, Meller S et al. Familial acute myeloid leukaemia: four male members of a single family over three consecutive generations exhibiting anticipation. Br J Haemat 1998; 100: 557–560.
4. Sellick GS, Pritchard-Jones K, Shepherd V, Swansbury J, Catovsky D, Houlston RS . Loci other than 21q22.12 (RUNX1) and 16q21–23.2 cause familial AML. Leukemia 2005; 19: 465–466.
5. Schwieger M, Lohler J, Fischer M, Herwig U, Tenen DG, Stocking C . A dominant-negative mutant of C/EBPalpha, associated with acute myeloid leukemias, inhibits differentiation of myeloid and erythroid progenitors of man but not mouse. Blood 2004; 103: 2744–2752.
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