Translocation t(1;19) is related to low cellular drug resistance in childhood acute lymphoblastic leukaemia-Reference-Cited by-同舟云学术

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Translocation t(1;19) is related to low cellular drug resistance in childhood acute lymphoblastic leukaemia

Published:2004-11-04 Issue:1 Volume:19 Page:165-169
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Frost B M, ,Forestier E,Gustafsson G,Nygren P,Hellebostad M,Jonmundsson G,Kanerva J,Schmiegelow K,Larsson R,Lönnerholm G

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2403540.pdf

Reference12 articles.

1. Rubnitz JE, Pui CH . Recent advances in the treatment and understanding of childhood acute lymphoblastic leukaemia. Cancer Treat Rev 2003; 29: 31–44.

2. Hunger SP . Chromosomal translocations involving the E2A gene in acute lymphoblastic leukemia: clinical features and molecular pathogenesis. Blood 1996; 87: 1211–1224.

3. Crist WM, Carroll AJ, Shuster JJ, Behm FG, Whitehead M, Vietti TJ et al. Poor prognosis of children with pre-B acute lymphoblastic leukemia is associated with the t(1;19)(q23;p13): a Pediatric Oncology Group study. Blood 1990; 76: 117–122.

4. Uckun FM, Sensel MG, Sather HN, Gaynon PS, Arthur DC, Lange BJ et al. Clinical significance of translocation t(1;19) in childhood acute lymphoblastic leukemia in the context of contemporary therapies: a report from the Children's Cancer Group. J Clin Oncol 1998; 16: 527–535.

5. Pui CH, Raimondi SC, Hancock ML, Rivera GK, Ribeiro RC, Mahmoud HH et al. Immunologic, cytogenetic, and clinical characterization of childhood acute lymphoblastic leukemia with the t(1;19) (q23; p13) or its derivative. J Clin Oncol 1994; 12: 2601–2606.

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