Expression and mutation status of candidate kinases in multiple myeloma-Reference-Cited by-同舟云学术

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Expression and mutation status of candidate kinases in multiple myeloma

Published:2007-03-08 Issue:5 Volume:21 Page:1124-1127
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Claudio J O,Zhan F,Zhuang L,Khaja R,Zhu Y X,Sivananthan K,Trudel S,Masih-Khan E,Fonseca R,Bergsagel P L,Scherer S W,Shaughnessy J,Stewart A K

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2404612.pdf

Reference5 articles.

1. Bardelli A, Parsons DW, Silliman N, Ptak J, Szabo S, Saha S et al. Mutational analysis of the tyrosine kinome in colorectal cancers. Science 2003; 300: 949.

2. Samuels Y, Wang Z, Bardelli A, Silliman N, Ptak J, Szabo S et al. High frequency of mutations of the PIK3CA gene in human cancers. Science 2004; 304: 554.

3. Oldridge M, Fortuna AM, Maringa M, Propping P, Mansour S, Pollitt C et al. Dominant mutations in ROR2,encoding an orphan receptor tyrosine kinase, cause brachydactyly type B. Nat Genet 2000; 24: 375–378.

4. Afzal AR, Rajab A, Fenske CD, Oldridge M, Elanko N, Ternes-Pereira E et al. Recessive Robinow syndrome, allelic to dominant brachydactyly type B, is caused by mutation of ROR2. Nat Genet 2000; 25: 419–422.

5. Billiard J, Way DS, Seestaller-Wehr LM, Moran RA, Mangine A, Bodine PV . The orphan receptor tyrosine kinase Ror2 modulates canonical Wnt signaling in osteoblastic cells. Mol Endocrinol 2005; 19: 90–101.

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