Recurrent chromosomal aberration at 12q15 in chronic idiopathic myelofibrosis with or without JAK2V617F mutation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/2404700.pdf
Reference8 articles.
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2. Schafer AI . Molecular basis of the diagnosis and treatment of polycythemia vera and essential thrombocythemia. Blood 2006; 107: 4214–4222.
3. Ohyashiki K, Aota Y, Akahane D, Gotoh A, Miyazawa K, Kimura Y et al. The JAK2 V617F tyrosine kinase mutation in myelodysplastic syndromes (MDS) developing myelofibrosis indicates the myeloproliferative nature in a subset of MDS patients. Leukemia 2005; 19: 2359–2360.
4. Kralovics R, Passamonti F, Buser AS, Teo SS, Tiedt R, Passweg JR et al. A gain-of-function mutation of JAK2 in myeloproliferative disorders. N Engl J Med 2005; 352: 1779–1790.
5. Nunoda K, Sashida G, Ohyashiki K, Kodama A, Fukutake K . The translocation (4;12)(q31;q21) in myelofibrosis associated with myelodysplastic syndrome: impact of the 12q21 breakpoint. Cancer Genet Cytogenet 2006; 164: 90–91.
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2. Chromosome 12 Rearrangement in an Adolescent with Primary Myelofibrosis;Indian Journal of Hematology and Blood Transfusion;2018-12-06
3. Increased likelihood of post-polycythemia vera myelofibrosis in Ph-negative MPN patients with chromosome 12 abnormalities;Leukemia Research;2015-04
4. The potential of carboxypeptidase M as a therapeutic target in cancer;Expert Opinion on Therapeutic Targets;2013-01-07
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