High expression of EDAG and its significance in AML-Reference-Cited by-同舟云学术

High expression of EDAG and its significance in AML

Published:2005-05-26 Issue:8 Volume:19 Page:1499-1502
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

An L-L,Li G,Wu K-F,Ma X-T,Zheng G-G,Qiu L-G,Song Y-H

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2403808.pdf

Reference8 articles.

1. Yang LV, Nicholson RH, Kaplan J, Galy A, Li L . Hemogen is a novel nuclear factor specifically expressed in mouse hematopoietic development and its human homologue EDAG maps to chromosome 9q22, a region containing breakpoints of hematological neoplasms. Mech Dev 2001; 104: 105–111.

2. Li CY, Zhan YQ, Xu CW, Xu WX, Wang SY, Lv J et al. EDAG regulates the proliferation and differentiation of hematopoietic cells and resists cell apoptosis through the activation of nuclear factor-kappaB. Cell Death Differ 2004; 11: 1299–1308.

3. Zhang B, Wang Y, Zheng GG, Ma XT, Li G, Zhang FK et al. Clinical significance of IL-18 gene over-expression in AML. Leukemia Res 2002; 26: 887–892.

4. Yang H, Wang SW, Yin HJ . p21(WAF1) gene transfection inhibits proliferation of leukemia cell line K562. Zhongguo Shi Yan Xue Ye Xue Za Zhi 2001; 9: 115–118.

5. Introna M, Golay J . How can oncogenic transcription factors cause cancer: a critical review of the myb story. Leukemia 1999; 13: 1301–1306.

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