Reply to Stams et al
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/2403577.pdf
Reference5 articles.
1. Stams WA, den Boer ML, Beverloo HB, van Wering ER, Pieters R . Upregulation of asparagine synthetase and cell cycle arrest in t(12;21) positive ALL. Leukemia 2005; 19: 318–319.
2. Krejci O, Starkova J, Otova B, Madzo J, Kalinova M, Hrusak O et al. Upregulation of asparagine synthetase fails to avert cell cycle arrest induced by L-asparaginase in TEL/AML1-positive leukaemic cells. Leukemia 2004; 18: 434–441.
3. Pui CH, Relling MV, Downing JR . Acute lymphoblastic leukemia. N Engl J Med 2004; 350: 1535–1548.
4. Andreasson P, Johansson B, Billstrom R, Garwicz S, Mitelman F, Hoglund M . Fluorescence in situ hybridization analyses of hematologic malignancies reveal frequent cytogenetically unrecognized 12p rearrangements. Leukemia 1998; 12: 390–400.
5. Kanerva J, Niini T, Vettenranta K, Riikonen P, Makipernaa A, Karhu R et al. Loss at 12p detected by comparative genomic hybridization (CGH): association with TEL-AML1 fusion and favorable prognostic features in childhood acute lymphoblastic leukemia (ALL). A multi-institutional study. Med Pediatr Oncol 2001; 37: 419–425.
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1. Expression levels of asparagine synthetase in blasts from children and adults with acute lymphoblastic leukaemia;British Journal of Haematology;2006-03
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