PIP3 controls synaptic function by maintaining AMPA receptor clustering at the postsynaptic membrane-Reference-Cited by-同舟云学术

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PIP3 controls synaptic function by maintaining AMPA receptor clustering at the postsynaptic membrane

Published:2009-12-13 Issue:1 Volume:13 Page:36-44
ISSN:1097-6256
Container-title:Nature Neuroscience
language:en
Short-container-title:Nat Neurosci

Author:

Arendt Kristin L,Royo María,Fernández-Monreal Mónica,Knafo Shira,Petrok Cortney N,Martens Jeffrey R,Esteban José A

Publisher

Springer Science and Business Media LLC

Subject

General Neuroscience

Link

http://www.nature.com/articles/nn.2462.pdf

Reference48 articles.

1. Cantley, L.C. The phosphoinositide 3-kinase pathway. Science 296, 1655–1657 (2002).

2. Di Paolo, G. & De Camilli, P. Phosphoinositides in cell regulation and membrane dynamics. Nature 443, 651–657 (2006).

3. Wenk, M.R. & De Camilli, P. Protein-lipid interactions and phosphoinositide metabolism in membrane traffic: insights from vesicle recycling in nerve terminals. Proc. Natl. Acad. Sci. USA 101, 8262–8269 (2004).

4. Sanna, P.P. et al. Phosphatidylinositol 3-kinase is required for the expression but not for the induction or the maintenance of long-term potentiation in the hippocampal CA1 region. J. Neurosci. 22, 3359–3365 (2002).

5. Opazo, P., Watabe, A.M., Grant, S.G. & O'Dell, T.J. Phosphatidylinositol 3-kinase regulates the induction of long-term potentiation through extracellular signal-related kinase-independent mechanisms. J. Neurosci. 23, 3679–3688 (2003).

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