Vav3 regulates osteoclast function and bone mass-Reference-Cited by-同舟云学术

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Vav3 regulates osteoclast function and bone mass

Published:2005-02-13 Issue:3 Volume:11 Page:284-290
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Faccio Roberta,Teitelbaum Steven L,Fujikawa Keiko,Chappel Jean,Zallone Alberta,Tybulewicz Victor L,Ross F Patrick,Swat Wojciech

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1194.pdf

Reference50 articles.

1. Schmidt, A. & Hall, A. Guanine nucleotide exchange factors for Rho GTPases: turning on the switch. Genes Dev. 16, 1587–1609 (2002).

2. Bustelo, X.R. Vav proteins, adaptors and cell signaling. Oncogene 20, 6372–6381 (2001).

3. Turner, M. & Billadeau, D.D. VAV proteins as signal integrators for multi-subunit immune-recognition receptors. Nat. Rev. Immunol. 2, 476–486 (2002).

4. Holsinger, L.J. et al. Defects in actin-cap formation in Vav-deficient mice implicate an actin requirement for lymphocyte signal transduction. Curr. Biol. 8, 563–572 (1998).

5. Fischer, K.D. et al. Vav is a regulator of cytoskeletal reorganization mediated by the T-cell receptor. Curr. Biol. 8, 554–562 (1998).

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