Excessive early-life dietary exposure: a potential source of elevated brain iron and a risk factor for Parkinson’s disease-Reference-Cited by-同舟云学术

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Excessive early-life dietary exposure: a potential source of elevated brain iron and a risk factor for Parkinson’s disease

Published:2017-01-05 Issue:1 Volume:3 Page:
ISSN:2373-8057
Container-title:npj Parkinson's Disease
language:en
Short-container-title:npj Parkinson's Disease

Author:

Hare Dominic J,Cardoso Bárbara Rita,Raven Erika P,Double Kay L,Finkelstein David I,Szymlek-Gay Ewa A,Biggs Beverley-Ann

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Neurology

Link

http://www.nature.com/articles/s41531-016-0004-y.pdf

Reference56 articles.

1. WHO. Guideline: Daily Iron Supplementation in Infants and Children (World Health Organization, 2016).

2. Li, W. et al. Differential developmental trajectories of magnetic susceptibility in human brain gray and white matter over the lifespan. Hum. Brain Mapp. 35, 2698–2713 (2013).

3. Ward, R. R., Zucca, F. A., Duyn, J. H., Crichton, R. R. & Zecca, L. The role of iron in brain ageing and neurodegenerativedisorders. Lancet Neurol. 13, 1045–1060 (2014).

4. Kaur, D., Rajagopalan, S. & Andersen, J. K. Chronic expression of H-ferritin in dopaminergic midbrain neurons results in an age-related expansion of the labile iron pool and subsequent neurodegeneration: implications for Parkinson’s disease. Brain Res. 1297, 17–22 (2009).

5. Hare, D. J. & Double, K. L. Iron and dopamine: a toxic couple. Brain 139, 1026–1035 (2016).

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