Nicastrin modulates presenilin-mediated notch/glp-1 signal transduction and βAPP processing-Reference-Cited by-同舟云学术

Nicastrin modulates presenilin-mediated notch/glp-1 signal transduction and βAPP processing

Published:2000-09 Issue:6800 Volume:407 Page:48-54
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Yu Gang,Nishimura Masaki,Arawaka Shigeki,Levitan Diane,Zhang Lili,Tandon Anurag,Song You-Qiang,Rogaeva Ekaterina,Chen Fusheng,Kawarai Toshitaka,Supala Agnes,Levesque Lyne,Yu Haung,Yang Dun-Sheng,Holmes Erin,Milman Paul,Liang Yan,Zhang Dong Mei,Xu Dong Hong,Sato Christine,Rogaev Evgeny,Smith Marsha,Janus Christopher,Zhang Yanni,Aebersold Ruedi,Farrer Lindsay,Sorbi Sandro,Bruni Amalia,Fraser Paul,St George-Hyslop Peter

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/35024009.pdf

Reference44 articles.

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2. Rogaev, E. I. et al. Familial Alzheimer's disease in kindreds with missense mutations in a novel gene on chromosome 1 related to the Alzheimer's disease type 3 gene. Nature 376, 775–778 (1995).

3. Yu, G. et al. The presenilin 1 protein is a component of a high molecular weight intracellular complex that contains β-catenin. J. Biol. Chem. 273, 16470–16475 ( 1998).

4. Nishimura, M. et al. Presenilin mutations dominantly modulate β-catenin trafficking. Nature Med. 5, 164–169 (1999).

5. De Strooper, B. et al. Post-translational modification, subcellular localization and membrane orientation of the Alzheimer's Disease associated Presenilins. J. Biol. Chem 272, 3590– 3598 (1997).

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