Colorectal cancer and genetic alterations in the Wnt pathway
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/1210059.pdf
Reference44 articles.
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2. Al-Tassan N, Chmiel NH, Maynard J, Fleming N, Livingston AL, Williams GT et al. (2002). Inherited variants of MYH associated with somatic G:C → T:A mutations in colorectal tumors. Nat Genet 30: 227–232.
3. Alazzouzi H, Davalos V, Kokko A, Domingo E, Woerner SM, Wilson AJ et al. (2005). Mechanisms of inactivation of the receptor tyrosine kinase EPHB2 in colorectal tumors. Cancer Res 65: 10170–10173.
4. Albuquerque C, Breukel C, van der Luijt R, Fidalgo P, Lage P, Slors FJ et al. (2002). The ‘just-right’ signaling model: APC somatic mutations are selected based on a specific level of activation of the beta-catenin signaling cascade. Hum Mol Genet 11: 1549–1560.
5. Batra S, Shi Y, Kuchenbecker KM, He B, Reguart N, Mikami I et al. (2006). Wnt inhibitory factor-1, a Wnt antagonist, is silenced by promoter hypermethylation in malignant pleural mesothelioma. Biochem Biophys Res Commun 342: 1228–1232.
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