Oncogene-induced replication stress preferentially targets common fragile sites in preneoplastic lesions. A genome-wide study-Reference-Cited by-同舟云学术

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Oncogene-induced replication stress preferentially targets common fragile sites in preneoplastic lesions. A genome-wide study

Published:2007-12-17 Issue:23 Volume:27 Page:3256-3264
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Tsantoulis P K,Kotsinas A,Sfikakis P P,Evangelou K,Sideridou M,Levy B,Mo L,Kittas C,Wu X-R,Papavassiliou A G,Gorgoulis V G

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1210989.pdf

Reference35 articles.

1. Aqeilan RI, Trapasso F, Hussain S, Costinean S, Marshall D, Pekarsky Y et al. (2007). Targeted deletion of Wwox reveals a tumor suppressor function. Proc Natl Acad Sci USA 104: 3949–3954.

2. Arlt MF, Durkin SG, Ragland RL, Glover TW . (2006). Common fragile sites as targets for chromosome rearrangements. DNA Repair (Amst) 5: 1126–1135.

3. Bartkova J, Hoesí Z, Koed K, Krämer A, Tort F, Zieger K et al. (2005). DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis. Nature 434: 864–870.

4. Bartkova J, Rezaei N, Liontos M, Karakaidos P, Kletsas D, Issaeva N et al. (2006). Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints. Nature 444: 633–637.

5. Batzer MA, Deininger PL . (2002). Alu repeats and human genomic diversity. Nat Rev Genet 3: 370–379.

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