p53 homologue, p51/p63, maintains the immaturity of keratinocyte stem cells by inhibiting Notch1 activity-Reference-Cited by-同舟云学术

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p53 homologue, p51/p63, maintains the immaturity of keratinocyte stem cells by inhibiting Notch1 activity

Published:2007-01-22 Issue:31 Volume:26 Page:4478-4488
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Okuyama R,Ogawa E,Nagoshi H,Yabuki M,Kurihara A,Terui T,Aiba S,Obinata M,Tagami H,Ikawa S

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1210235.pdf

Reference43 articles.

1. Artavanis-Tsakonas S, Rand MD, Lake RJ . (1999). Notch signaling: cell fate control and signal integration in development. Science 284: 770–776.

2. Bourdon JC, Fernandes K, Murray-Zmijewski F, Liu G, Diot A, Xirodimas DP et al. (2005). p53 isoforms can regulate p53 transcriptional activity. Genes Dev 19: 2122–2137.

3. Celli J, Duijf P, Hamel BC, Bamshad M, Kramer B, Smits AP et al. (1999). Heterozygous germline mutations in the p53 homolog p63 are the cause of EEC syndrome. Cell 99: 143–153.

4. Crook T, Nicholls JM, Brooks L, O'Nions J, Allday MJ . (2000). High level expression of deltaN-p63: a mechanism for the inactivation of p53 in undifferentiated nasopharyngeal carcinoma (NPC)? Oncogene 19: 3439–3444.

5. Cui R, He J, Mei R, de Fromentel CC, Martel-Planche G, Taniere P et al. (2005). Expression of p53, p63, and p73 isoforms in squamous cell carcinoma and adenocarcinoma of esophagus. Biochem Biophys Res Commun (in press).

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