P53 in blind subterranean mole rats – loss-of-function versus gain-of-function activities on newly cloned Spalax target genes-Reference-Cited by-同舟云学术

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P53 in blind subterranean mole rats – loss-of-function versus gain-of-function activities on newly cloned Spalax target genes

Published:2006-10-16 Issue:17 Volume:26 Page:2507-2512
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Avivi A,Ashur-Fabian O,Joel A,Trakhtenbrot L,Adamsky K,Goldstein I,Amariglio N,Rechavi G,Nevo E

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1210045.pdf

Reference33 articles.

1. Ashur-Fabian O, Avivi A, Trakhtenbrot L, Adamsky K, Cohen M, Kajakaro G et al. (2004). Evolution of p53 in hypoxia-stressed Spalax mimics human tumor mutation. Proc Natl Acad Sci USA 101: 12236–12241.

2. Avivi A, Ashur-Fabian O, Amariglio N, Nevo E, Rechavi G . (2005a). p53 – a key player in tumoral and evolutionary adaptation: a lesson from the Israeli blind subterranean mole rat. Cell Cycle 4: 368–372.

3. Avivi A, Resnick MB, Nevo E, Joel A, Levy AP . (1999). Adaptive hypoxic tolerance in the subterranean mole rat Spalax ehrenbergi: the role of vascular endothelial growth factor. FEBS Lett 452: 133–140.

4. Avivi A, Shams I, Joel A, Lache O, Levy AP, Nevo E . (2005b). Increased blood vessel density provides the mole rat physiological tolerance to its hypoxic subterranean habitat. FASEB J 19: 1314–1316.

5. el-Deiry WS, Kern SE, Pietenpol JA, Kinzler KW, Vogelstein B . (1992). Definition of a consensus binding site for p53. Nat Genet 1: 45–49.

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