Conditional expression of mutated K-ras accelerates intestinal tumorigenesis in Msh2-deficient mice

Author:

Luo F,Brooks D G,Ye H,Hamoudi R,Poulogiannis G,Patek C E,Winton D J,Arends M J

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Reference44 articles.

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3. Arends MJ, Frayling I . (2004). The genetics of colorectal cancer: mismatch repair deficiency in hereditary and sporadic colorectal cancer. In: Cunningham D, Topham C, Miles A (eds). Key Advances in the Effective Management of Colorectal Cancer Chapter 2, 4th edn., Aesculapius Medical Press: London, pp 25–40.

4. Arends MJ, McGregor AH, Toft NJ, Brown EJ, Wyllie AH . (1993). Susceptibility to apoptosis is differentially regulated by c-myc and mutated Ha-ras oncogenes and is associated with endonuclease availability. Br J Cancer 68: 1127–1133.

5. Arends MJ, McGregor AH, Wyllie AH . (1994). Apoptosis is inversely related to necrosis and determines net growth in tumours bearing constitutively expressed myc, ras, and HPV oncogenes. Am J Pathol 144: 1045–1057.

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