Comparative expression profiling identifies an in vivo target gene signature with TFAP2B as a mediator of the survival function of PAX3/FKHR

Author:

Ebauer M,Wachtel M,Niggli F K,Schäfer B W

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Reference28 articles.

1. Anderson J, Ramsay A, Gould S, Pritchard-Jones K . (2001). PAX3-FKHR induces morphological change and enhances cellular proliferation and invasion in rhabdomyosarcoma. Am J Pathol 159: 1089–1096.

2. Ayyanathan K, Fredericks WJ, Berking C, Herlyn M, Balakrishnan C, Gunther E et al. (2000). Hormone-dependent tumor regression in vivo by an inducible transcriptional repressor directed at the PAX3-FKHR oncogene. Cancer Res 60: 5803–5814.

3. Begum S, Emani N, Cheung A, Wilkins O, Der S, Hamel PA . (2005). Cell-type-specific regulation of distinct sets of gene targets by Pax3 and PAX3/FKHR. Oncogene 24: 1860–1872.

4. Bennicelli JL, Fredericks WJ, Wilson RB, Rauscher III FJ, Barr FG . (1995). Wild type PAX3 protein and the PAX3-FKHR fusion protein of alveolar rhabdomyosarcoma contain potent, structurally distinct transcriptional activation domains. Oncogene 11: 119–130.

5. Bernasconi M, Remppis A, Fredericks WJ, Rauscher III FJ, Schafer BW . (1996). Induction of apoptosis in rhabdomyosarcoma cells through down-regulation of PAX proteins. Proc Natl Acad Sci USA 93: 13164–13169.

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