Phosphorylation by Akt disables the anti-oncogenic activity of YB-1
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/1210719.pdf
Reference19 articles.
1. Aoki M, Batista O, Bellacosa A, Tsichlis P, Vogt PK . (1998). The Akt kinase: molecular determinants of oncogenicity. Proc Natl Acad Sci USA 95: 14950–14955.
2. Aoki M, Blazek E, Vogt PK . (2001). A role of the kinase mTOR in cellular transformation induced by the oncoproteins P3k and Akt. Proc Natl Acad Sci USA 98: 136–141.
3. Aoki M, Schetter C, Himly M, Batista O, Chang HW, Vogt PK . (2000). The catalytic subunit of phosphoinositide 3-kinase: requirements for oncogenicity. J Biol Chem 275: 6267–6275.
4. Bader AG, Felts KA, Jiang N, Chang HW, Vogt PK . (2003). Y box-binding protein 1 induces resistance to oncogenic transformation by the phosphatidylinositol 3-kinase pathway. Proc Natl Acad Sci USA 100: 12384–12389.
5. Bader AG, Kang S, Zhao L, Vogt PK . (2005). Oncogenic PI3K deregulates transcription and translation. Nat Rev Cancer 5: 921–929.
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