FLJ10540-elicited cell transformation is through the activation of PI3-kinase/AKT pathway-Reference-Cited by-同舟云学术

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FLJ10540-elicited cell transformation is through the activation of PI3-kinase/AKT pathway

Published:2007-01-22 Issue:29 Volume:26 Page:4272-4283
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Chen C-H,Lu P-J,Chen Y-C,Fu S-L,Wu K-J,Tsou A-P,Lee Y-C G,Lin T-C E,Hsu S-L,Lin W-J,Huang C-Y F,Chou C-K

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1210207.pdf

Reference29 articles.

1. Bellacosa A, Chan TO, Ahmed NN, Datta K, Malstrom S, Stokoe D et al. (1998). Akt activation by growth factors is a multiple-step process: the role of the PH domain. Oncogene 17: 313–325.

2. Cantley LC . (2002). The phosphoinositide 3-kinase pathway. Science 296: 1655–1657.

3. Chen X, Cheung ST, So S, Fan ST, Barry C, Higgins J et al. (2002). Gene expression patterns in human liver cancers. Mol Biol Cell 13: 1929–1939.

4. Chen YL, Law PY, Loh HH . (2004). Inhibition of akt/protein kinase B signaling by naltrindole in small cell lung cancer cells. Cancer Res 64: 8723–8730.

5. Fabbro M, Zhou BB, Takahashi M, Sarcevic B, Lal P, Graham ME et al. (2005). Cdk1/Erk2- and Plk1-dependent phosphorylation of a centrosome protein, Cep55, is required for its recruitment to midbody and cytokinesis. Dev Cell 9: 477–488.

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