The transcriptional response after oxidative stress is defective in Cockayne syndrome group B cells-Reference-Cited by-同舟云学术

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The transcriptional response after oxidative stress is defective in Cockayne syndrome group B cells

Published:2003-02 Issue:8 Volume:22 Page:1135-1149
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Kyng Kasper J,May Alfred,Brosh Robert M,Cheng Wen-Hsing,Chen Catheryne,Becker Kevin G,Bohr Vilhelm A

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/1206187.pdf

Reference55 articles.

1. Allen RG and Tresini M . (2000). Oxidative stress and gene regulation. Free Radic. Biol. Med., 28, 463–499.

2. Balajee AS and Bohr VA . (2000). Genomic heterogeneity of nucleotide excision repair. Gene, 250, 15–30.

3. Balajee AS, May A, Dianov GL, Friedberg EC and Bohr VA . (1997). Reduced RNA polymerase II transcription in intact and permeabilized Cockayne syndrome group B cells. Proc. Natl. Acad. Sci. USA, 94, 4306–4311.

4. Bootcov MR, Bauskin AR, Valenzuela SM, Moore AG, Bansal M, He XY, Zhang HP, Donnellan M, Mahler S, Pryor K, Walsh BJ, Nicholson RC, Fairlie WD, Por SB, Robbins JM and Breit SN . (1997). MIC-1, a novel macrophage inhibitory cytokine, is a divergent member of the TGF-beta superfamily. Proc. Natl. Acad. Sci. USA, 94, 11514–11519.

5. Bregman DB, Halaban R, Van bool AJ, Henning KA, Friedberg EC and Warren SL . (1996). UV-induced ubiquitination of RNA polymerase II: a novel modification deficient in Cockayne syndrome cells. Proc. Natl. Acad Sci. USA, 93, 11586–11590.

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