HPV16 E6 confers p53-dependent and p53-independent phenotypes in the epidermis of mice deficient for E6AP
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cancer Research,Genetics,Molecular Biology
Link
http://www.nature.com/articles/1210130.pdf
Reference55 articles.
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3. Chellappan S, Kraus VB, Kroger B, Munger K, Howley PM, Phelps WC et al. (1992). Adenovirus E1A, simian virus 40 tumor antigen, and human papillomavirus E7 protein share the capacity to disrupt the interaction between transcription factor E2F and the retinoblastoma gene product. Proc Natl Acad Sci USA 89: 4549–4553.
4. Cooper B, Schneider S, Bohl J, Jiang Y, Beaudet A, Vande Pol S . (2003). Requirement of E6AP and the features of human papillomavirus E6 necessary to support degradation of p53. Virology 306: 87–99.
5. Dyson N, Howley PM, Munger K, Harlow E . (1989). The human papillomavirus-16 E7 oncoprotein is able to bind to the retinoblastoma gene product. Science 243: 934–937.
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