Glucocorticoid-resistant B cell acute lymphoblastic leukemia displays receptor tyrosine kinase activation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics (clinical),Genetics,Molecular Biology
Link
http://www.nature.com/articles/s41525-019-0082-y.pdf
Reference31 articles.
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2. Ratman, D. et al. How glucocorticoid receptors modulate the activity of other transcription factors: a scope beyond tethering. Mol. Cell Endocrinol. 380, 41–54 (2013).
3. Biddie, S. C. et al. Transcription factor AP1 potentiates chromatin accessibility and glucocorticoid receptor binding. Mol. Cell 43, 145–155 (2011).
4. Altonsy, M. O., Sasse, S. K., Phang, T. L. & Gerber, A. N. Context-dependent cooperation between nuclear factor kappaB (NF-kappaB) and the glucocorticoid receptor at a TNFAIP3 intronic enhancer: a mechanism to maintain negative feedback control of inflammation. J. Biol. Chem. 289, 8231–8239 (2014).
5. Rao, N. A. et al. Coactivation of GR and NFKB alters the repertoire of their binding sites and target genes. Genome Res 21, 1404–1416 (2011).
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