Human autoinflammatory disease reveals ELF4 as a transcriptional regulator of inflammation-Reference-Cited by-同舟云学术

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Human autoinflammatory disease reveals ELF4 as a transcriptional regulator of inflammation

Published:2021-07-29 Issue:9 Volume:22 Page:1118-1126
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Tyler Paul M.,Bucklin Molly L.^ORCID,Zhao Mengting,Maher Timothy J.,Rice Andrew J.,Ji Weizhen,Warner Neil^ORCID,Pan Jie,Morotti Raffaella,McCarthy Paul,Griffiths Anne,van Rossum Annemarie M. C.,Hollink Iris H.I.M.,Dalm Virgil A.S.H.,Catanzaro Jason^ORCID,Lakhani Saquib A.,Muise Aleixo M.^ORCID,Lucas Carrie L.^ORCID

Funder

U.S. Department of Health & Human Services | NIH | National Institute of Allergy and Infectious Diseases

G. Harold and Leila Y. Mathers Foundation

Charles H. Hood Foundation

Immune Deficiency Foundation

National Science Foundation

Canada Research Chairs

U.S. Department of Health & Human Services | NIH | National Institute of Diabetes and Digestive and Kidney Diseases

Leona M. and Harry B. Helmsley Charitable Trust

Canadian Institute of Health Research (CIHR) Foundation

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

https://www.nature.com/articles/s41590-021-00984-4.pdf

Reference45 articles.

1. Bousfiha, A. et al. Human inborn errors of immunity: 2019 update of the IUIS phenotypical classification. J. Clin. Immunol. 40, 66–81 (2020).

2. Tangye, S. G. et al. Human inborn errors of immunity: 2019 update on the classification from the International Union of Immunological Societies Expert Committee. J. Clin. Immunol. 40, 24–64 (2020).

3. Adzhubei, I. A. et al. A method and server for predicting damaging missense mutations. Nat. Methods 7, 248–249 (2010).

4. Karczewski, K. J. et al. The mutational constraint spectrum quantified from variation in 141,456 humans. Nature 581, 434–443 (2020).

5. Rentzsch, P., Witten, D., Cooper, G. M., Shendure, J. & Kircher, M. CADD: predicting the deleteriousness of variants throughout the human genome. Nucleic Acids Res. 47, D886–D894 (2019).

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