Adaptive response to inflammation contributes to sustained myelopoiesis and confers a competitive advantage in myelodysplastic syndrome HSCs-Reference-Cited by-同舟云学术

Adaptive response to inflammation contributes to sustained myelopoiesis and confers a competitive advantage in myelodysplastic syndrome HSCs

Published:2020-04-20 Issue:5 Volume:21 Page:535-545
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Muto Tomoya,Walker Callum S.,Choi Kwangmin,Hueneman Kathleen,Smith Molly A.,Gul Zartash,Garcia-Manero Guillermo,Ma Averil^ORCID,Zheng Yi,Starczynowski Daniel T.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/s41590-020-0663-z.pdf

Reference55 articles.

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3. Nilsson, L. et al. Isolation and characterization of hematopoietic progenitor/stem cells in 5q-deleted myelodysplastic syndromes: evidence for involvement at the hematopoietic stem cell level. Blood. 96, 2012–2021 (2000).

4. Muto, T. et al. Concurrent loss of Ezh2 and Tet2 cooperates in the pathogenesis of myelodysplastic disorders. J. Exp. Med. 210, 2627–2639 (2013).

5. Fang, J. et al. Ubiquitination of hnRNPA1 by TRAF6 links chronic innate immune signaling with myelodysplasia. Nat. Immunol. 18, 236–245 (2017).

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