c-Kit-positive ILC2s exhibit an ILC3-like signature that may contribute to IL-17-mediated pathologies-Reference-Cited by-同舟云学术

c-Kit-positive ILC2s exhibit an ILC3-like signature that may contribute to IL-17-mediated pathologies

Published:2019-07-01 Issue:8 Volume:20 Page:992-1003
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Bernink Jochem H.,Ohne Yoichiro,Teunissen Marcel B. M.^ORCID,Wang Jingya,Wu Jincheng,Krabbendam Lisette,Guntermann Christine^ORCID,Volckmann Richard,Koster Jan^ORCID,van Tol Sophie,Ramirez Ivan^ORCID,Shrestha Yashaswi^ORCID,de Rie Menno A.,Spits Hergen^ORCID,Romero Ros Xavier,Humbles Alison A.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/s41590-019-0423-0.pdf

Reference47 articles.

1. Artis, D. & Spits, H. The biology of innate lymphoid cells. Nature 517, 293–301 (2015).

2. Spits, H. et al. Innate lymphoid cells—a proposal for a uniform nomenclature. Nat. Rev. Immunol. 13, 145–149 (2013).

3. Bernink, J. H. et al. Human type 1 innate lymphoid cells accumulate in inflamed mucosal tissues. Nat. Immunol. 14, 221–229 (2013).

4. Bal, S. M. et al. IL-1beta, IL-4 and IL-12 control the fate of group 2 innate lymphoid cells in human airway inflammation in the lungs. Nat. Immunol. 17, 636–645 (2016).

5. Ohne, Y. et al. IL-1 is a critical regulator of group 2 innate lymphoid cell function and plasticity. Nat. Immunol. 17, 646–655 (2016).

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