Stimulation history is epigenetically imprinted in memory B cells
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Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41590-024-01903-z.pdf
Reference5 articles.
1. Dogan, I. et al. Multiple layers of B cell memory with different effector functions. Nat. Immunol. 10, 1292–1299 (2009). A research article providing initial evidence for the association of memory B cell isotype with fate preferences.
2. Zuccarino-Catania, G. V. et al. CD80 and PD-L2 define functionally distinct memory B cell subsets that are independent of antibody isotype. Nat. Immunol. 15, 631–637 (2014). A research article that reports subsets of memory B cells defined by CD80 and PD-L2 exhibit different fate preferences.
3. Sciammas, R. et al. An incoherent regulatory network architecture that orchestrates B cell diversification in response to antigen signaling. Mol. Syst. Biol. 7, 495 (2011). A research article that proposes a kinetic model for how the regulatory network of transcription factors affects B cell differentiation in response to antigen signaling.
4. Weisel, F. J., Zuccarino-Catania, G. V., Chikina, M. & Shlomchik, M. J. A temporal switch in the germinal center determines differential output of memory B and plasma cells. Immunity 44, 116–130 (2016). A research article that uses a pulse-labeling experiment to show that later GCs produce more plasma cells than memory B cells.
5. Mesin, L. et al. Restricted clonality and limited germinal center reentry characterize memory B cell reactivation by boosting. Cell 180, 92–106 (2020). A research article that shows that memory B cells seldomly seed secondary GCs.
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