A dietary cholesterol by-product regulates intestinal IgA secretion via GPR183
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://www.nature.com/articles/s41590-023-01442-z.pdf
Reference5 articles.
1. Altmann, S. W. et al. Niemann-Pick C1 Like 1 protein is critical for intestinal cholesterol absorption. Science 303, 1201–1204 (2004). This paper shows that NPC1L1 is required for intestinal absorption of dietary cholesterol.
2. Russell, D. W. The enzymes, regulation, and genetics of bile acid synthesis. Annu. Rev. Biochem. 72, 137–174 (2003). A review article that summarizes the biochemical requirements for cholesterol metabolite generation.
3. Frascoli, M., Reboldi, A. & Kang, J. Dietary cholesterol metabolite regulation of tissue immune cell development and function. J. Immunol. 209, 645–653 (2022). A review article describing the crosstalk between oxysterols and immune cells.
4. Hannedouche, S. et al. Oxysterols direct immune cell migration via EBI2. Nature 475, 524–527 (2011). This paper identifies 7α,25-hydroxycholesterol as the most potent ligand of GPR183.
5. Kelly, L. M., Pereira, J. P., Yi, T., Xu, Y. & Cyster, J. G. EBI2 guides serial movements of activated B cells and ligand activity is detectable in lymphoid and nonlymphoid tissues. J. Immunol. 187, 3026–3032 (2011). This paper describes an in vitro bioassay to measure GPR183 ligand in tissues.
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