Bcl11b sets pro-T cell fate by site-specific cofactor recruitment and by repressing Id2 and Zbtb16-Reference-Cited by-同舟云学术

Bcl11b sets pro-T cell fate by site-specific cofactor recruitment and by repressing Id2 and Zbtb16

Published:2018-10-30 Issue:12 Volume:19 Page:1427-1440
ISSN:1529-2908
Container-title:Nature Immunology
language:en
Short-container-title:Nat Immunol

Author:

Hosokawa Hiroyuki^ORCID,Romero-Wolf Maile,Yui Mary A.^ORCID,Ungerbäck Jonas,Quiloan Maria L. G.,Matsumoto Masaki,Nakayama Keiichi I.,Tanaka Tomoaki^ORCID,Rothenberg Ellen V.^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/s41590-018-0238-4.pdf

Reference70 articles.

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2. Liu, P., Li, P. & Burke, S. Critical roles of Bcl11b in T-cell development and maintenance of T-cell identity. Immunol. Rev. 238, 138–149 (2010).

3. Shibata, K. et al. IFN-γ-producing and IL-17-producing γδ T cells differentiate at distinct developmental stages in murine fetal thymus. J. Immunol. 192, 2210–2218 (2014).

4. Kueh, H. Y. et al. Asynchronous combinatorial action of four regulatory factors activates Bcl11b for T cell commitment. Nat. Immunol. 17, 956–965 (2016).

5. Ikawa, T. et al. An essential developmental checkpoint for production of the T cell lineage. Science 329, 93–96 (2010).

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