Migration-induced cell shattering due to DOCK8 deficiency causes a type 2–biased helper T cell response
Author:
Publisher
Springer Science and Business Media LLC
Subject
Immunology,Immunology and Allergy
Link
https://www.nature.com/articles/s41590-020-0795-1.pdf
Reference64 articles.
1. Aydin, S. E. et al. DOCK8 deficiency: clinical and immunological phenotype and treatment options - a review of 136 patients. J. Clin. Immunol. 35, 189–198 (2015).
2. Su, H. C., Jing, H., Angelus, P. & Freeman, A. F. Insights into immunity from clinical and basic science studies of DOCK8 immunodeficiency syndrome. Immunol. Rev. 287, 9–19 (2019).
3. Cote, J. F. Identification of an evolutionarily conserved superfamily of DOCK180-related proteins with guanine nucleotide exchange activity. J. Cell Sci. 115, 4901–4913 (2002).
4. Zhang, Q. et al. DOCK8 regulates lymphocyte shape integrity for skin antiviral immunity. J. Exp. Med. 211, 2549–2566 (2014).
5. Tangye, S. G. et al. Dedicator of cytokinesis 8-deficient CD4+ T cells are biased to a TH2 effector fate at the expense of TH1 and TH17 cells. J. Allergy Clin. Immunol. 139, 933–949 (2017).
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