Low nitric oxide bioavailability contributes to the genesis of experimental cerebral malaria-Reference-Cited by-同舟云学术

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Low nitric oxide bioavailability contributes to the genesis of experimental cerebral malaria

Published:2006-11-12 Issue:12 Volume:12 Page:1417-1422
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Gramaglia Irene,Sobolewski Peter,Meays Diana,Contreras Ramiro,Nolan John P,Frangos John A,Intaglietta Marcos,van der Heyde Henri C

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1499.pdf

Reference30 articles.

1. Stevenson, M.M. & Riley, E.M. Innate immunity to malaria. Nat. Rev. Immunol. 4, 169–180 (2004).

2. Taylor-Robinson, A.W., Phillips, R.S., Severn, A., Moncada, S. & Liew, F.Y. The role of TH1 and TH2 cells in a rodent malaria infection. Science 260, 1931–1934 (1993).

3. Clark, I.A. & Cowden, W.B. The pathophysiology of falciparum malaria. Pharmacol. Ther. 99, 221–260 (2003).

4. Chang, W.L. et al. CD8+-T-cell depletion ameliorates circulatory shock in Plasmodium berghei–infected mice. Infect. Immun. 69, 7341–7348 (2001).

5. Combes, V., De Souza, J.B., Renia, L., Hunt, N.H. & Grau, G.E. Cerebral malaria: Which parasite? Which model? Drug Discovery Today 2, 141–147 (2005).

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