ATRAP in the paraventricular nucleus of the hypothalamus as another key player in the control of sympathetic outflow
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Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41440-024-01602-0.pdf
Reference12 articles.
1. Katsurada K, Ogoyama Y, Imai Y, Patel KP, Kario K. Renal denervation based on experimental rationale. Hypertens Res. 2021;44:1385–94.
2. Llewellyn T, Zheng H, Liu X, Xu B, Patel KP. Median preoptic nucleus and subfornical organ drive renal sympathetic nerve activity via a glutamatergic mechanism within the paraventricular nucleus. Am J Physiol Regul Integr Comp Physiol. 2012;302:R424–432.
3. Llewellyn TL, Sharma NM, Zheng H, Patel KP. Effects of exercise training on SFO-mediated sympathoexcitation during chronic heart failure. Am J Physiol Heart Circ Physiol. 2014;306:H121–131.
4. Sharma NM, Haibara AS, Katsurada K, Nandi SS, Liu X, Zheng H, et al. Central ang II (angiotensin II)-mediated sympathoexcitation: role for HIF-1alpha (hypoxia-inducible factor-1alpha) aacilitated glutamatergic tone in the paraventricular nucleus of the hypothalamus. Hypertension. 2021;77:147–57.
5. Li YF, Wang W, Mayhan WG, Patel KP. Angiotensin-mediated increase in renal sympathetic nerve discharge within the PVN: role of nitric oxide. Am J Physiol Regul Integr Comp Physiol. 2006;290:R1035–1043.
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