Relations between glomerular hyperfiltration and podocyte injury: potential role of Piezo1 in the Rac1-mineralocorticoid receptor activation pathway
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Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s41440-024-01603-z.pdf
Reference8 articles.
1. Kopp JB, Anders HJ, Susztak K, Podesta MA, Remuzzi G, Hildebrandt F, et al. Podocytopathies. Nat Rev Dis Prim. 2020;6:68.
2. Lucero CM, Prieto-Villalobos J, Marambio-Ruiz L, Balmazabal J, Alvear TF, Vega M, et al. Hypertensive nephropathy: unveiling the possible involvement of hemichannels and pannexons. Int J Mol Sci. 2022;23:15936.
3. Fukuda A, Minakawa A, Kikuchi M, Sato Y, Nagatomo M, Nakamura S, et al. Urinary podocyte mRNAs precede microalbuminuria as a progression risk marker in human type 2 diabetic nephropathy. Sci Rep. 2020;10:18209.
4. Wu J, Lewis AH, Grandl J. Touch, tension, and transduction—the function and regulation of Piezo ion channels. Trends Biochem Sci. 2017;42:57–71.
5. Shibata S, Nagase M, Yoshida S, Kawarazaki W, Kurihara H, Tanaka H, et al. Modification of mineralocorticoid receptor function by Rac1 GTPase: implication in proteinuric kidney disease. Nat Med. 2008;14:1370–6.
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