Cardioprotection by direct factor Xa inhibition in angiotensin II overexpression
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cardiology and Cardiovascular Medicine,Physiology,Internal Medicine
Link
https://www.nature.com/articles/s41440-021-00721-2.pdf
Reference9 articles.
1. Ebrahimi S, Rezaei S, Seiri P, Ryzhikov M, Hashemy SI, Hassanian SM. Factor Xa signaling contributes to the pathogenesis of inflammatory diseases. J Cell Physiol. 2017;232:1966–70.
2. Macfarlane SR, Seatter MJ, Kanke T, Hunter GD, Plevin R. Proteinase-activated receptors. Pharm Rev. 2001;53:245–82.
3. Ichikawa H, Shimada M, Narita M, Narita I, Kimura Y, Tanaka M, et al. Rivaroxaban, a direct factor Xa inhibitor, ameliorates hypertensive renal damage through inhibition of the inflammatory response mediated by protease-activated receptor pathway. J Am Heart Assoc. 2019;8:e012195.
4. Bode MF, Auriemma AC, Grover SP, Hisada Y, Rennie A, Bode WD, et al. The factor Xa inhibitor rivaroxaban reduces cardiac dysfunction in a mouse model of myocardial infarction. Thromb Res. 2018;167:128–34.
5. Nakanishi N, Kaikita K, Ishii M, Oimatsu Y, Mitsuse T, Ito M, et al. Cardioprotective effects of rivaroxaban on cardiac remodeling after experimental myocardial infarction in mice. Circ Rep. 2020;2:158–66.
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