High levels of GDF15 in thalassemia suppress expression of the iron regulatory protein hepcidin-Reference-Cited by-同舟云学术

High levels of GDF15 in thalassemia suppress expression of the iron regulatory protein hepcidin

Published:2007-08-26 Issue:9 Volume:13 Page:1096-1101
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Tanno Toshihiko,Bhanu Natarajan V,Oneal Patricia A,Goh Sung-Ho,Staker Pamela,Lee Y Terry,Moroney John W,Reed Christopher H,Luban Naomi LC,Wang Rui-Hong,Eling Thomas E,Childs Richard,Ganz Tomas,Leitman Susan F,Fucharoen Suthat,Miller Jeffery L

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm1629.pdf

Reference30 articles.

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2. Pootrakul, P. et al. A correlation of erythrokinetics, ineffective erythropoiesis, and erythroid precursor apoptosis in Thai patients with thalassemia. Blood 96, 2606–2612 (2000).

3. Nathan, D.G. Thalassemia as a proliferative disorder. Medicine (Baltimore) 43, 779–782 (1964).

4. Weatherall, D.J. & Clegg, J.B. The Thalassaemia Syndromes (4th ed.) (Blackwell Scientific, Oxford, 2001).

5. Nemeth, E. & Ganz, T. Regulation of iron metabolism by hepcidin. Annu. Rev. Nutr. 26, 323–342 (2006).

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