A pathway of osmotic stress-induced necroptosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Nephrology
Link
https://www.nature.com/articles/s41581-022-00607-7.pdf
Reference10 articles.
1. Linkermann, A., Stockwell, B. R., Krautwald, S. & Anders, H. J. Regulated cell death and inflammation: an auto-amplification loop causes organ failure. Nat. Rev. Immunol. 14, 759–767 (2014).
2. Zhang, W., Fan, W., Guo, J. & Wang, X. Osmotic stress activates RIPK3/MLKL-mediated necroptosis by increasing cytosolic pH through a plasma membrane Na+/H+ exchanger. Sci. Signal. 15, eabn5881 (2022).
3. Wakabayashi, S. et al. Overexpression of Na+/H+ exchanger 1 specifically induces cell death in human iPS cells via sustained activation of the Rho kinase ROCK. J. Biol. Chem. 294, 19577–19588 (2019).
4. Sharifi, M. et al. Necroptosis and RhoA/ROCK pathways: molecular targets of Nesfatin-1 in cardioprotection against myocardial ischemia/reperfusion injury in a rat model. Mol. Biol. Rep. 48, 2507–2518 (2021).
5. Martin-Sanchez, D. et al. Bone marrow-derived RIPK3 mediates kidney inflammation in acute kidney injury. J. Am. Soc. Nephrol. 33, 357–373 (2022).
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